Search results for "Immunity to Infections"

showing 8 items of 8 documents

Keratinocytes Determine Th1 Immunity during Early Experimental Leishmaniasis

2010

Experimental leishmaniasis is an excellent model system for analyzing Th1/Th2 differentiation. Resistance to Leishmania (L.) major depends on the development of a L. major specific Th1 response, while Th2 differentiation results in susceptibility. There is growing evidence that the microenvironment of the early affected tissue delivers the initial triggers for Th-cell differentiation. To analyze this we studied differential gene expression in infected skin of resistant and susceptible mice 16h after parasite inoculation. Employing microarray technology, bioinformatics, laser-microdissection and in-situ-hybridization we found that the epidermis was the major source of immunomodulatory mediat…

KeratinocytesCellular differentiationImmunology/Innate ImmunityInterleukin-1betaGene ExpressionInfectious Diseases/Skin InfectionsMiceT-Lymphocyte SubsetsLeishmania majorBiology (General)In Situ HybridizationOligonucleotide Array Sequence AnalysisSkinRegulation of gene expressionMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionCell DifferentiationImmunohistochemistryInterleukin-12MicrodissectionResearch ArticleQH301-705.5ImmunologyLeishmaniasis CutaneousBiologyMicrobiologyTh2 CellsImmune systemCutaneous leishmaniasisImmunology/Immunity to InfectionsVirologyGeneticsmedicineAnimalsDermatology/Skin InfectionsMolecular BiologyInterleukin 4Epidermis (botany)Interleukin-6Gene Expression ProfilingLasersTh1 CellsRC581-607medicine.diseasebiology.organism_classificationMice Inbred C57BLGene expression profilingDisease Models AnimalImmunology/Immune ResponseImmunologyOsteopontinParasitologyInterleukin-4Immunologic diseases. AllergyPLoS Pathogens
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Expression of Toll-Like Receptors in the Developing Brain

2012

Toll-like receptors (TLR) are key players of the innate and adaptive immune response in vertebrates. The original protein Toll in Drosophila melanogaster regulates both host defense and morphogenesis during development. Making use of real-time PCR, in situ hybridization, and immunohistochemistry we systematically examined the expression of TLR1-9 and the intracellular adaptor molecules MyD88 and TRIF during development of the mouse brain. Expression of TLR7 and TLR9 in the brain was strongly regulated during different embryonic, postnatal, and adult stages. In contrast, expression of TLR1-6, TLR8, MyD88, and TRIF mRNA displayed no significant changes in the different phases of brain develop…

AgingGene Expressionlcsh:MedicineMiceMolecular Cell BiologyMorphogenesislcsh:ScienceReceptorImmune ResponseRegulation of gene expressionMultidisciplinaryNeocortexToll-Like ReceptorsBrainGene Expression Regulation DevelopmentalAcquired immune systemInnate ImmunityCell biologyInfectious Diseasesmedicine.anatomical_structureMedicineResearch ArticleImmunologyCentral nervous systemMorphogenesisIn situ hybridizationBiologyMolecular GeneticsImmune ActivationDevelopmental NeuroscienceGeneticsmedicineAnimalsHumansRNA MessengerBiologyImmunity to Infectionslcsh:RImmunityComputational BiologyImmune DefenseAxonsHEK293 CellsTRIFImmune SystemCellular NeuroscienceImmunologyClinical Immunologylcsh:QTranscriptomeDevelopmental BiologyNeurosciencePLoS ONE
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Validation of the Tetracycline Regulatable Gene Expression System for the Study of the Pathogenesis of Infectious Disease

2011

Understanding the pathogenesis of infectious disease requires the examination and successful integration of parameters related to both microbial virulence and host responses. As a practical and powerful method to control microbial gene expression, including in vivo, the tetracycline-regulatable system has recently gained the favor of many investigative groups. However, some immunomodulatory effects of the tetracyclines, including doxycycline, could potentially limit its use to evaluate host responses during infection. Here we have used a well-established murine model of disseminated candidiasis, which is highly dependent on both the virulence displayed by the fungal cells and on the host im…

ChemokineScienceImmunologyVirulenceMycologyPathogenesisKidneyResponse ElementsMicrobiologyMicrobiologyPathogenesisMiceGene Expression Regulation FungalCandida albicansGene expressionmedicineAnimalsPromoter Regions GeneticCandida albicansBiologyImmunity to InfectionsProtein Synthesis InhibitorsDoxycyclineMultidisciplinaryVirulencebiologyQCandidiasisImmunityRTetracyclinebiology.organism_classificationDisseminated CandidiasisDisease Models AnimalInfectious DiseasesMedical MicrobiologyInfectious disease (medical specialty)DoxycyclineHost-Pathogen InteractionsMutationImmunologybiology.proteinCytokinesMedicineChemokinesSpleenResearch Articlemedicine.drug
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Virulent synergistic effect between Enterococcus faecalis and Escherichia coli assayed by using the Caenorhabditis elegans model.

2008

5 pages; International audience; BACKGROUND: The role of enterococci in the pathogenesis of polymicrobial infections is still debated. The purpose of this study was to evaluate the effect of virulent enterococci in the presence or absence of Escherichia coli strains in the in vivo Caenorhabditis elegans model. PRINCIPAL FINDINGS: This study demonstrated that there was a synergistic effect on virulence when an association of enterococci and E. coli (LT50 = 1.6 days+/-0.1 according to the tested strains and death of nematodes in 4 days+/-0.5) was tested in comparison with enterococci alone (LT50 = 4.6 days+/-0.1 and death in 10.4 days+/-0.6) or E. coli alone (LT50 = 2.1+/-0.9 and deaths 6.6+/…

MESH : Virulence FactorsInfectious Diseases/Gastrointestinal InfectionsMESH : Escherichia colilcsh:MedicineMESH : Genotypemedicine.disease_causeMESH: Regression AnalysisPathogenesisMESH: GenotypeInfectious Diseases/Bacterial InfectionsMESH : Regression AnalysisGenotype[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisEnterococcus faecalis[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: AnimalsMESH : Anti-Bacterial AgentsMESH : Enterococcus faecalislcsh:ScienceCaenorhabditis elegans0303 health sciencesMultidisciplinarybiologyMESH: Escherichia coliBacterial Infections3. Good healthAnti-Bacterial AgentsMicrobiology/Immunity to InfectionsMESH : Bacterial InfectionsGastroenterology and Hepatology/Gastrointestinal Infections[SDV.IMM]Life Sciences [q-bio]/ImmunologyRegression AnalysisMicrobiology/Cellular Microbiology and PathogenesisResearch ArticleMESH: Enterococcus faecalis[SDV.IMM] Life Sciences [q-bio]/ImmunologyGenotypeMESH: Bacterial InfectionsVirulence FactorsVirulenceEnterococcus faecalisMicrobiologyMESH : Caenorhabditis elegans03 medical and health sciencesIn vivoMESH: Anti-Bacterial AgentsMESH: Caenorhabditis elegansmedicineEscherichia coliAnimalsCaenorhabditis elegansEscherichia coli030304 developmental biologyMESH: Virulence Factors030306 microbiologylcsh:RMicrobiology/Medical Microbiology[SDV.EE.IEO] Life Sciences [q-bio]/Ecology environment/Symbiosisbiology.organism_classificationMESH : Disease Models AnimalDisease Models AnimalEnterococcuslcsh:QMESH : AnimalsMESH: Disease Models Animal[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisPloS one
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Increase in gut microbiota after immune suppression in baculovirus-infected larvae.

2013

Spodoptera exigua microarray was used to determine genes differentially expressed in S. exigua cells challenged with the species-specific baculovirus SeMNPV as well as with a generalist baculovirus, AcMNPV. Microarray results revealed that, in contrast to the host transcriptional shut-off that is expected during baculovirus infection, S. exigua cells showed a balanced number of up- and down-regulated genes during the first 36 hours following the infection. Many immune-related genes, including pattern recognition proteins, genes involved in signalling and immune pathways as well as immune effectors and genes coding for proteins involved in the melanization cascade were found to be down-regul…

MicroarraysApplied MicrobiologyvirusesGut floraTranscriptomesBiology (General)Immune ResponseEffectorViral Immune EvasionMicrobiotaAgricultureGenomicsFunctional GenomicsHost-Pathogen InteractionIntestinesLarvaResearch ArticleQH301-705.5Mechanisms of Resistance and SusceptibilityImmunologyVirulenceBiologySpodopteraSpodopteraImmune SuppressionMicrobiologydigestive systemVirusMicrobiologyMolecular GeneticsImmune systemIntegrated ControlGenome Analysis ToolsVirologyMicrobial ControlExiguaGeneticsImmune ToleranceAnimalsGene RegulationMolecular BiologyGeneBiologyImmunity to InfectionsMicrobial PathogensImmunityComputational BiologyImmune DefenseRC581-607biology.organism_classificationNucleopolyhedrovirusesParasitologyPest ControlImmunologic diseases. AllergyGenome Expression AnalysisPLoS Pathogens
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Chlamydia trachomatis Infection and Anti-Hsp60 Immunity: The Two Sides of the Coin

2009

Chlamydia trachomatis (CT) infection is one of the most common causes of reproductive tract diseases and infertility. CT-Hsp60 is synthesized during infection and is released in the bloodstream. As a consequence, immune cells will produce anti-CT-Hsp60 antibodies. Hsp60, a ubiquitous and evolutionarily conserved chaperonin, is normally sequestered inside the cell, particularly into mitochondria. However, upon cell stress, as well as during carcinogenesis, the chaperonin becomes exposed on the cell surface (sf-Hsp60) and/or is secreted from cells into the extracellular space and circulation. Reports in the literature on circulating Hsp and anti-Hsp antibodies are in many cases short on detai…

lcsh:Immunologic diseases. Allergyanimal structuresImmunologyCardiovascular Disorders/Heart FailurePublic Health and Epidemiology/Infectious DiseasesChlamydia trachomatisPathology/Immunologychemical and pharmacologic phenomenaReviewmedicine.disease_causecomplex mixturesMicrobiologyAutoimmune DiseasesInfectious Diseases/Bacterial InfectionsPathogenesisImmune systemImmunityVirologyGeneticsmedicineAnimalsHumansImmunology/Cellular Microbiology and Pathogenesislcsh:QH301-705.5Molecular BiologyRheumatology/Autoimmunity Autoimmune and Inflammatory DiseasesAntigens BacterialbiologySettore BIO/16 - Anatomia UmanaMultiple sclerosisfungiAutoantibodyChaperonin 60Chlamydia Infectionsmedicine.diseaseHSP60 ChlamydiaMicrobiology/Immunity to Infectionslcsh:Biology (General)Immunologybiology.proteinParasitologyHSP60AntibodyDiabetes and Endocrinology/Type 1 Diabeteslcsh:RC581-607Chlamydia trachomatisPLoS Pathogens
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Immune Modulating Effects of NKT Cells in a Physiologically Low Dose Leishmania major Infection Model after αGalCer Analog PBS57 Stimulation

2014

Leishmaniasis is a parasitic infection affecting ∼12 million people worldwide, mostly in developing countries. Treatment options are limited and no effective vaccines exist to date. Natural Killer T (NKT) cells are a conserved innate-like lymphocyte population with immunomodulating effects in various settings. A number of reports state a role of NKT cells in different models of Leishmania infection. Here, we investigated the effect of NKT cells in a physiologically relevant, intradermal low dose infection model. After inoculation of 103 infectious-stage L. major, comparable numbers of skin-immigrating NKT cells in both susceptible BALB/c mice and resistant C57BL/6 mice were noted. Compared …

medicine.medical_treatmentLymphocyteMedizinPathogenesisNK cellsProtozoologyPathology and Laboratory MedicineCellular typesMedicine and Health SciencesLymphoid OrgansLeishmania majorImmune ResponseLeishmania majorSkinProtozoansMice Inbred BALB Ceducation.field_of_studybiologylcsh:Public aspects of medicineNatural killer T cellInfectious Diseasesmedicine.anatomical_structureCytokineMedical MicrobiologyHost-Pathogen InteractionsWhite blood cellsCytokinesAnatomyResearch ArticleCell biologyBlood cellslcsh:Arctic medicine. Tropical medicinelcsh:RC955-962Immune CellsImmunologyPopulationT cellsLeishmaniasis CutaneousGalactosylceramidesSpleenImmunopathologyMicrobiologyLymphatic SystemImmunomodulationImmune ActivationImmune systemImmunityMicrobial ControlmedicineAnimalsImmunologic FactorseducationImmunity to InfectionsMicrobial PathogensBiology and life sciencesImmunityOrganismsPublic Health Environmental and Occupational HealthImmunoregulationlcsh:RA1-1270Molecular Developmentbiology.organism_classificationAcquired Immune SystemParasitic ProtozoansMice Inbred C57BLDisease Models AnimalAnimal cellsImmune SystemImmunologyNatural Killer T-CellsClinical ImmunologyParasitologyDevelopmental Biology
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Analysis of Mycobacterium tuberculosis-Specific CD8 T-Cells in Patients with Active Tuberculosis and in Individuals with Latent Infection

2009

CD8 T-cells contribute to control of Mycobacterium tuberculosis infection, but little is known about the quality of the CD8 T-cell response in subjects with latent infection and in patients with active tuberculosis disease. CD8 T-cells recognizing epitopes from 6 different proteins of Mycobacterium tuberculosis were detected by tetramer staining. Intracellular cytokines staining for specific production of IFN-gamma and IL-2 was performed, complemented by phenotyping of memory markers on antigen-specific CD8 T-cells. The ex-vivo frequencies of tetramer-specific CD8 T-cells in tuberculous patients before therapy were lower than in subjects with latent infection, but increased at four months a…

MaleEpitopes T-Lymphocytelcsh:MedicineCD8-Positive T-LymphocytesEpitopeDiagnostic RadiologyInfectious Diseases/Bacterial InfectionsSpectrum Analysis TechniquesCellular typesCytotoxic T celllcsh:ScienceImage Cytometryeducation.field_of_studyMultidisciplinarybiologyRadiology and ImagingImmune cellsInfection ImagingMiddle AgedFlow CytometryActinobacteriaPhenotypeSpectrophotometryCytokinesWhite blood cellsFemaleCytophotometryResearch Articlemedicine.drugInterleukin 2Cell biologyBlood cellsTuberculosisImaging TechniquesImmunologyPopulationT cellsCytotoxic T cellsResearch and Analysis MethodsMycobacterium tuberculosisDiagnostic MedicineImmunology/Immunity to InfectionsHLA-A2 AntigenmedicineHumansTuberculosiseducationMedicine and health sciencesHLA-A AntigensBacteriaFluorimetrylcsh:ROrganismsBiology and Life SciencesMycobacterium tuberculosisbiology.organism_classificationmedicine.diseaseVirologyRetractionAnimal cellsImmunology/Immune ResponseImmunologyMycobacterium tuberculosis CD8 T cells Tuberculosis Latent Infectionlcsh:QCD8MycobacteriumPLoS ONE
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